@misc{cogprints4298, volume = {2}, number = {4}, month = {October}, author = {Yuji Nakazato}, editor = {Balbir Singh and Yash Lokhandwala and Johnson Francis and Anup Gupta}, title = {Tachycardiomyopathy }, publisher = {Indian Pacing and Electrophysiology Group}, year = {2002}, journal = {Indian Pacing and Electrophysiology Journal}, pages = {104--113}, keywords = {Tachycardiomyopathy}, url = {http://cogprints.org/4298/}, abstract = {Sustained chronic tachyarrhythmias often cause a deterioration of cardiac function known as tachycardia-induced cardiomyopathy or tachycardiomyopathy.1 It has been recognized that the tachycardiomyopathy occurs in experimental models2 and also in patients with supraventricular or ventricular tachycarrhythmias.3,4,5,6 Generally, cardiac function will recover if drug or catheter ablation therapy is successfully performed. Phillips and Levine7 initially presented this concept in 1949 for the relationship between rapid atrial fibrillation and reversible left ventricular (LV) failure. However, it might be difficult to define its cause and effect relationship when cardiomyopathy and tachycardia are identified simultaneously. The reversibility of LV dysfunction may often be variable and the precise mechanisms of the pathophysiological features of this phenomenon should be elucidated. Although excellent and comprehensive review articles by Fenelon G, et al.8 and Shinbane JS et al 9 regarding these topics have already been published, I will firstly present a typical case and then review the basic and clinical characteristics of tachycardiomyopathy in this article. } }